Autonomic tone
Controls that mostly shape sinus rate, respiratory coupling, and baroreflex-style variability.
Neurocardiac ECG Lab
Read the ECG as a brain-heart interface
The surface tracing is now framed through autonomic tone, respiratory coupling, and AV nodal braking. Instead of only tweaking voltages, you can move through realistic vagal and sympathetic teaching states, then inspect how those shifts alter the ECG paper, rhythm strip, and cardiac vector.
Active preset
Balanced Rest
API target
internal Next.js route handlers
Autonomic presets
Shift the tracing through realistic physiological states
Choose a preset, then fine-tune the grouped controls below.
Display toggles
ECG-paper realism
Paper speed
Lead layout
Faster paper speeds show fewer milliseconds across the same screen width, which makes interval inspection feel much closer to bedside ECG review.
Consult frames
Read the strip through neurocardiac scenarios
These frames push the lab beyond parameter twiddling. Pick the consult scenario, load the aligned physiology, and then decide what the strip is really saying versus what it could be mistaken for.
Syndrome frame
Acute neurogenic stress pattern
The tracing is fast, relatively fixed, and catecholamine-weighted. The teaching question is whether the ECG is reflecting primary cardiac disease or an autonomic response to acute CNS injury or severe stress physiology.
Highest-yield next data
- • Serial troponins and dynamic ECG trend rather than one tracing in isolation
- • Bedside correlation with the neurological trigger, echo pattern, and hemodynamic context
Compare mode
Best mechanism versus attractive misread
Best mechanism
Central sympathetic surge with neurocardiac spillover, producing a tachycardic and repolarization-shifted surface pattern.
- • Neurogenic ECG changes are often diagnosis-adjacent but not diagnosis-complete.
- • The wrong move is to let the strip erase the neurological context that generated it.
Weaker alternative
Primary ACS by ECG appearance alone
An isolated tracing cannot outrank the physiology. When sympathetic context is overwhelming, repolarization distortion can be secondary and demands correlation rather than reflexively becoming the diagnosis.
Conduction timing
Intervals that change AV nodal delay, depolarization width, and repolarization timing.
Morphology and acquisition
Amplitude and projection controls that shape what the surface leads record and how strongly they appear.
3D cardiac activation
Dipole replay through the beat
Lead constellation
Which lead sees the strongest vector
Representative beat phase
Scrub through activation timing
Generate ECG to animate a representative beat.
Surface ECG
Clinical 12-lead sheet
Showing 6000 ms at 25 mm/s with a 10 mm/mV teaching reference.