Vestibular Localization

Vertigo and vestibular localizer

Teach dizziness through timing, triggers, hearing clues, gait burden, and the eye-movement exam. The goal is to separate canal, labyrinth, vestibular-nerve, stroke, and migraine-network syndromes without collapsing every spinning complaint into one bucket.

Current frame: Peripheral acute vestibular syndrome

Syndrome presets

Start from the bedside vertigo grammar

Students remember vertigo best when you organize it by temporal pattern: continuous acute vestibular syndrome, brief triggered spells, recurrent cochlear episodes, and visually loaded migraine attacks.

Oculomotor trace

Eye-movement pattern under the current syndrome

slow phase drift plus quick resettimeintensity envelope

Unidirectional horizontal-torsional nystagmus that intensifies when gazing in the fast-phase direction.

Duration ladder

Timing usually beats buzzwords

secminhrdayweek

Continuous for 1 to 3 days before compensation starts easing it. Usually one prolonged attack rather than many tiny bursts.

Gaze loading

Does the beat direction stay loyal?

left gaze88%
primary62%
right gaze34%

Peripheral syndromes often intensify in one gaze direction. Central syndromes change direction or lose that clean pattern.

Provocation map

What makes the vertigo declare itself

rest72%
head turn92%
positional34%
visual motion48%

Timing and triggers are often more localizing than the word dizziness itself.

Phenotype

Vestibular neuritis

central risk 16%head impulse positive

Continuous hours-to-days vertigo with nausea, unidirectional horizontal-torsional nystagmus, an abnormal head impulse, and no meaningful cochlear symptoms.

Central risk

16%

Not a diagnostic score, but a teaching shorthand for how hard the bedside data pull you toward brainstem-cerebellar causes.

Fixation suppression

78%

Peripheral nystagmus tends to calm when the patient fixates. Central patterns usually ignore that brake.

Gait burden

58%

The more catastrophic the truncal instability, the more you should worry about posterior fossa localization.

Hearing shift

6%

Cochlear symptoms are major localization clues in labyrinthine disease and selected AICA lesions.

Vestibular pathway

Where the lesion pressure sits

posteriorcanallabyrinthnervenucleicerebellumcortexvestibular signal path from ear to brain

Peripheral vestibular apparatus, usually the superior vestibular nerve on one side.

HINTS readout

What the exam is really saying

Head impulse

Positive to the injured side with a corrective catch-up saccade.

Nystagmus

Unidirectional horizontal-torsional nystagmus that intensifies when gazing in the fast-phase direction.

Test of skew

Absent.

HINTS is appropriate here because the syndrome is continuous acute vestibular syndrome, not a brief positional spell.

One labyrinth suddenly under-fires, so the brain interprets the asymmetry as ongoing head rotation. That creates a slow-phase drift toward the injured side and quick phases away from it.

Differential pivots

What should win and what should lose

A corrective saccade on head impulse plus unidirectional nystagmus and absent skew is classic peripheral acute vestibular syndrome.

Posterior circulation stroke becomes weaker when HINTS stays peripheral and hearing is preserved, but central red flags still win if the gait is severely truncal or focal deficits appear.

Trigger pattern

Head turns worsen the illusion, but the patient is still dizzy at rest.

Hearing frame

Hearing is typically preserved, which separates it from cochlear syndromes.

Bedside maneuvers

Examinations that actually settle the syndrome

HINTS exam

Positive head impulse, unidirectional nystagmus, and no skew pull the syndrome toward the ear rather than the brainstem.

This is the pattern where HINTS helps you avoid reflexively overcalling stroke in a continuously dizzy patient.

Hearing screen

Finger rub and tuning-fork testing are usually symmetric.

Spared hearing supports vestibular neuritis over labyrinthitis or AICA territory ischemia.

Gait check

The patient feels awful and may veer, but can usually still stand or walk with support.

Catastrophic truncal collapse is more worrisome for cerebellar stroke than for isolated neuritis.

Supportive teaching

How to frame the syndrome for learners and patients

  • Keep the first explanation simple: one inner ear is under-signaling, so the brain thinks the head is still moving.
  • After the hyperacute phase, encourage gentle head movement and visual fixation tasks so central compensation can start.
  • Avoid reinforcing bed rest longer than necessary once the dangerous central mimics are excluded.
A positive head impulse in acute continuous vertigo is reassuringly peripheral. A normal head impulse in the same syndrome is the dangerous result.

Continue the loop

Pair vestibular reasoning with anatomy, stroke, gait, and tutoring

Cranial Nerves Explorer

Clinical neurological examination

Stroke Vascular Territories

Acute neurovascular localization

Gait Pattern Localizer

Bedside gait localization

Brain Atlas

Post-clinical anatomical convergence

Neuro Tutor

Cross-module consult reasoning with explicit scoring